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<br> Schmitt CA, Fridman JS, Yang M, Baranov E, Hoffman RM, Lowe SW (April 2002). "Dissecting p53 tumor suppressor functions in vivo". Arsic N, Gadea G, Lagerqvist EL, Busson M, Cahuzac N, Brock C, Hollande F, Gire V, Pannequin J, Roux P (April 2015). "The p53 isoform Δ133p53β promotes cancer stem cell potential". Khoury MP, Bourdon JC (April 2011). "p53 Isoforms: An Intracellular Microprocessor?". Surget S, Khoury MP, Bourdon JC (December 2013). "Uncovering the part of p53 splice variants in human malignancy: a scientific standpoint". Thurow HS, Haack R, Hartwig FP, Oliveira IO, Dellagostin OA, Gigante DP, Horta BL, Collares T, Seixas FK (December 2011). "TP53 gene polymorphism: relevance to most cancers, ethnicity and start body weight in a Brazilian cohort". Wang JJ, Zheng Y,  [https://Streempornfree.com/tag/top-trans-pornstars/ Top-trans-Pornstars] Sun L, Wang L, Yu PB, Dong JH, Zhang L, Xu J, Shi W, Ren YC (November 2011). "TP53 codon 72 polymorphism and colorectal most cancers susceptibility: a meta-examination". Wang XW, Tseng A, Ellis NA, Spillare EA, Linke SP, Robles AI, Seker H, Yang Q, Hu P, Beresten S, Bemmels NA, Garfield S, Harris CC (August 2001). "Functional conversation of p53 and BLM DNA helicase in apoptosis".<br><br><br><br> Yang Q, Zhang R, Wang XW, Spillare EA, Linke SP, Subramanian D, Griffith JD, Li JL, Hickson ID, Shen JC, Loeb LA, Mazur SJ, Appella E, Brosh RM, Karmakar P, Bohr VA, Harris CC (August 2002). "The processing of Holliday junctions by BLM and WRN helicases is controlled by p53". Canner JA, Sobo M, Ball S, Hutzen B, DeAngelis S, Willis W, Studebaker AW, Ding K, Wang S, Yang D, Lin J (September 2009). "MI-63: a novel smaller-molecule inhibitor targets MDM2 and induces apoptosis in embryonal and alveolar rhabdomyosarcoma cells with wild-style p53". Marión RM, Strati K, Li H, Murga M, Blanco R, Ortega S, Fernandez-Capetillo O, Serrano M, Blasco MA (August 2009). "A p53-mediated DNA harm reaction restrictions reprogramming to assure iPS cell genomic integrity". Venot C, Maratrat M, Dureuil C, Conseiller E, Bracco L, Debussche L (August 1998). "The necessity for the p53 proline-rich functional area for mediation of apoptosis is correlated with distinct PIG3 gene transactivation and with transcriptional repression". Han JM, Park BJ, Park SG, Oh YS, Choi SJ, Lee SW, Hwang SK, Chang SH, Cho MH, Kim S (August 2008). "AIMP2/p38, the scaffold for the multi-tRNA synthetase complicated, responds to genotoxic stresses via p53". Tyner SD, Venkatachalam S, Choi J, Jones S, Ghebranious N, Igelmann H, Lu X, Soron G, Cooper B, Brayton C, Park SH, Thompson T, Karsenty G, Bradley A, Donehower LA (January 2002). "p53 mutant mice that show early ageing-related phenotypes".<br><br><br><br> Pearson S, Jia H, Kandachi K (January 2004). "China approves initially gene therapy". Kojic S, Medeot E, Guccione E, Krmac H, Zara I, Martinelli V, Valle G, Faulkner G (May 2004). "The Ankrd2 protein, a url involving the sarcomere and the nucleus in skeletal muscle mass". Leu JI, Dumont P, Hafey M, Murphy ME, George DL (May 2004). "Mitochondrial p53 activates Bak and will cause disruption of a Bak-Mcl1 intricate". Toufektchan E, Toledo F (May 2018). "The Guardian of the Genome Revisited: p53 Downregulates Genes Required for Telomere Maintenance, DNA Repair, and Centromere Structure". Westphal CH, Schmaltz C, Rowan S, Elson A, Fisher DE, Leder P (May 1997). "Genetic interactions concerning atm and p53 affect mobile proliferation and irradiation-induced cell cycle checkpoints". Ko LJ, Shieh SY, Chen X, Jayaraman L, Tamai K, Taya Y, Prives C, Pan ZQ (December 1997). "p53 is phosphorylated by CDK7-cyclin H in a p36MAT1-dependent fashion". Dong Y, Hakimi MA, Chen X, Kumaraswamy E, Cooch NS, Godwin AK, Shiekhattar R (November 2003). "Regulation of BRCC, a holoenzyme sophisticated made up of BRCA1 and BRCA2, by a signalosome-like subunit and its role in DNA repair". Harms KL, Chen X (March 2005). "The C terminus of p53 spouse and children proteins is a cell fate determinant".<br><br><br><br> AG, SINGULUS Technologies (March 3, 2020). "CD Production Replication Machine". Mitkin NA, Hook CD, Schwartz AM, Biswas S, Kochetkov DV, Muratova AM, Afanasyeva MA, Kravchenko JE, Bhattacharyya A, Kuprash DV (March 2015). "p53-dependent expression of CXCR5 chemokine receptor in MCF-7 breast most cancers cells". Assadian S, El-Assaad W, Wang XQ, Gannon PO, Barrès V, Latour M, Mes-Masson AM, Saad F, Sado Y, Dostie J, Teodoro JG (March 2012). "p53 inhibits angiogenesis by inducing the manufacturing of Arresten". Yu H, Huang YJ, Liu Z, Wang LE, Li G, Sturgis EM, Johnson DG, Wei Q (September 2011). "Effects of MDM2 promoter polymorphisms and p53 codon seventy two polymorphism on danger and age at onset of squamous cell carcinoma of the head and neck". Sonoyama T, Sakai A, Mita Y, Yasuda Y, Kawamoto H, Yagi T, Yoshioka M, Mimura T, Nakachi K, Ouchida M, Yamamoto K, Shimizu K (2011). "TP53 codon seventy two polymorphism is involved with pancreatic most cancers danger in males, people who smoke and drinkers". Due to the isoformic nature of p53 proteins, there have been many resources of proof showing that mutations within just the TP53 gene providing rise to mutated isoforms are causative agents of many cancer phenotypes, from moderate to critical, because of to solitary mutation in the TP53 gene (refer to section Experimental evaluation of p53 mutations for extra information).<br>